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Research data from Princeton University on a genetic element in C. elegans composed of the sperm-delivered toxin peel-1 and the embryo-expressed antidote zeel-1. The study demonstrates how this element causes late-stage embryo lethality and gains a transmission advantage. The dataset likely contains experimental results on gene expression, protein function, and transmission distortion.
License is listed as Open Access (green), but specific terms should be verified from the source paper.